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Several studies using HCC cell lines suggests that ferroptosis may serve as the principal mechanism underlying the anticancer effect of sorafenib via suppressing the cystine/glutamate antiporter SLC7A11 [94,95,96]

10.1021/acsnano.8b06266 6 CaiY.XuZ.ShuaiQ.ZhuF.XuJ.GaoX.et al (2020)

they actively modulate localized immune responses

Cytokines such as IL-1 and TNF- also upregulate ACSL4 expression in neurons, promoting PUFA oxidation and enhancing lipid peroxidation, which increases neuronal susceptibility to ferroptosis