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Biochemical reactions rely heavily on electron transfer mediated by NAD + /NADH and NADP + /NADPH couples, which not only serve as cofactors but also act as substrates in enzymatic reactions, sustaining redox homeostasis and cellular energetics

In contrast, mutation of the C-terminal active site cysteine increases the activity of GLRX, presumably by preventing disulfide bond formation between the two active site cysteines 20,21 , and renders GLRX a monothiol enzyme only capable of catalysis of (de)glutathionylation 22

Increases in p53 expression induce CTGF synthesis by mouse and human hepatocytes and result in liver fibrosis in mice

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