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Case studies and systematic reviews, such as those by Fitzpatrick et al

demonstrated that GPx8 also covalently associates with the conserved residue in caspase-4 (C118), but not murine or human caspase-1, to inhibit caspase-4 activation in response to intracellular LPS ( Gpx8 and higher CASP4 expression ratios in the colon when compared to controls suggesting that GPx8 protects against coilitis by acting as a negative regulator of caspase-4/-11 ( In agreement with the above findings, genome-wide association studies have identified antioxidant genes, including GPX4, as IBD susceptibility loci ( Gpx4 is incompatible with life and results in early embryonic lethality, illustrating its importance ( t al