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Y1H, biotin-IP and ChIP-qPCR showed that GCR1/2 were able to bind directly to the LFS promoter (Fig

GSTM1/GSTT1 backstop both simultaneously when each is impaired and the absence of all three defines the most compromised Phase 2 architecture in the panel

The inhibition of intracellular antioxidant status such as GSH suggests that F-induced oxidative stress is a potential mechanism underlying the toxic effects of F on reproductive organs [63]