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Preventing this decrease, with the administration of a SIRT-1 activator, reversed ROS bursts, increased downstream nuclear PGC-1a and Nrf2 expression, and attenuated increases in mitochondrial membrane potential and neuronal apoptosis induced by ischemia/reperfusion injury [78]

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Further studies are warranted to elucidate the mechanisms by which GMSC-derived Schwann-like cells modulate pro-inflammatory (M1)/pro-regenerative (M2) macrophage polarization in the setting of PNI

The HSV-1 VP5 signal remained undetectable during the first 60 min of infection, and then gradually increased at later time points