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9 Conclusion and future prospects The pathogenesis of ALD is inextricably linked to dysregulation of OS-related signaling networks, involving dynamic interactions among key molecules such as SREBP, Nrf2, and AMPK

Scientists studying metabolic pathways and NAD+ homeostasis have recognized 5-Amino-1MQ as a valuable probe for understanding how NNMT inhibition affects cellular function

Insulin-like growth factors (IGFs) signaling through the phosphatidylinositol 3-kinase (PI3K/Akt) downstream pathway can protect neurons against LPS excitotoxicity in cell culture and [124, 128,129]