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Description

Although data are not available to date, this study is one of the first to address the link between genetic susceptibility and response to treatment in SLE

Epstein-Barr virus lacking glycoprotein gp42 can bind to B cells but is not able to infect

Regulatory mechanisms in GBM pathogenesis Epigenetic characteristics of GBM GBM pathogenesis is driven by a combination of extensive genetic and epigenetic alterations that regulate gene expression and tumor progression