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Selenium reduction of ubiquinone via SQOR suppresses ferroptosis

Hormonal alterations Since stress has been described as a potential trigger for ME/CFS (422) and cause of symptom flare-ups (423, 424), it is likely that the HPA axis- the neurobiological stress system- may be implicated in ME/CFS (53, 425, 426), as well as abnormalities in growth hormone (GH) secretion and dysfunctional adrenergic metabolism (255)

These reversible, regulatory oxiPTMs on specific cysteines have emerged as important mechanisms that alter protein function post-translationally 1,2,6,10,11,12

These data can be interpreted as there is a positive relationship between disease severity and oxidative stress severity in OCD