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Moreover, blocking the SAB peptide prevents palmitic acid-induced P-JNK interaction with SAB and inhibition of mitochondrial bioenergetics, implying the P-JNK effect on mitochondrial metabolism

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One of the reasons for high failure rates in CNS drug development is limited access of drugs to the target site in the brain

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Cumins therapeutic potential for toothaches, epilepsy, and gastrointestinal maladies has been discussed the most (Mnif and Aifa, 2015)

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