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2a) and steric blockage mechanisms (Fig

A better understanding of the structurefunction relationships of glutaredoxins has therefore numerous physiological and even biotechnological implications

Third, binding of transition metal ion catalysts to prevent generation of initiating ROS

Results from the present study further corresponded well with this viewpoint in which increment of Fe 2+ expression coincided well with the up-regulation of ATF6, IRE1, PERK, and BAX activities (Figures 5, 6), all are key elements participated in the development of ER stress and apoptosis