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When mitochondrial function is impaired, decreased ATP levels lead to increased TrkB internalization, forming a vicious cycle in which mitochondrial dysfunction causes impaired NTFs signal reception

Significantly, a major challenge currently lies in the selective targeting of lactylation without affecting other PTMs, as they are intricately linked and share a common set of regulatory enzymes, such as p300, HDACs, and Sirts, complicating the specific inhibition of lactylation

In a 3-week study, Szepietowski et al

Therefore, abnormal iron metabolism aggravates cellular oxidative stress, induces ferroptosis, and promotes the triggering of AF