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The wound-healing potential of novel tripeptides GHK-AgNPs and GHK-Cu-AgNPs was assessed both in vitro using mouse dermal fibroblasts (PAM212 and L929) and in vivo in S

SCF/ -TrCP promotes glycogen synthase kinase 3-dependent degradation of the Nrf2 transcription factor in a Keap1-independent manner

Analysis of signaling pathways that underlie microglial activation and production of proinflammatory mediators showed that benfotiamine suppressed activation of ERK1/2 and JNK members of MAPK signaling pathway, as well as protein kinase B/Akt

The specimens were subsequently de-mineralized in 10% ethylene-diamine-tetraacetic acid (EDTA) solution (pH = 7.4) for 4 weeks then embedded in paraffin