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Therefore, the implication from this work is that a large portion of the S in normal cellular steady-state GSH pools is derived from Met via the Met-cycle and transsulfuration

Intestine-specific FTN-deficiency leads to marked increases in intestinal iron absorption and inducing iron overload, and an unidentified novel microbial metabolite regulates systemic FTN homeostasis (Das et al., 2020)

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