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note that ROS effects depend on the specific target cell, our framework operationalizes this by identifying the specific gap in antioxidant capacitywhere tumor cells and Tregs are metabolically rewired to tolerate oxidative pressure that is lethal to Teffsas the active driver of immune evasion (Table 1)

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In Kombination mit Calciumphosphat-Scaffolds beschleunigte GHK-Cu die Remineralisierung von Dentin und die Proliferation von Odontoblasten