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Glutamine metabolism inhibition, using agents such as CB-839, has shown potential to suppress TCA cycle-mediated energy production and disrupt redox homeostasis, particularly in CRC subtypes resistant to 5-FU and oxaliplatin [145]

Adv Enzyme Regul 22:2755 Coen M (2015) Metabolic phenotyping applied to pre-clinical and clinical studies of acetaminophen metabolism and hepatotoxicity

Patients with extensive brain damage, severe cognitive impairment, or other critical health issues might not be good candidates for treatment

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