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A mouse model of amyloid beta oligomers: their contribution to synaptic alteration, abnormal tau phosphorylation, glial activation, and neuronal loss in vivo

These factors are known to impede the maturation and functionality of DCs within the TME (216)

Preconditioning with quercetin in normoxia group does not lead to any alterations in any of these transcriptional factors and their associated proteins in comparison with normoxia control group