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Findings from early translational studies showed that the CPT I inhibitor, Etomoxir, had a cardioprotective role in the ischaemic rat heart by decreasing cellular LCAC content and promoting glycolytic substrate utilization ( Limitations in the Literature A limitation of the LCAC literature is that all research was performed using non-human models of the mammalian myocardium

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It is well known that the free radicals lead to oxidative stress, which causes damage to DNA, protein, lipid peroxidation, and cell membrane damage