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Additionally, damage to the descending lateral corticospinal tract at T1 causes the observed spasticity and hyperreflexia in the lower extremities, while disruption of the ascending spinothalamic tract explains the loss of pinprick sensation

Zhang J, Lazarenko OP, Kang J, et al

Science 337, 11011104

When HEESCs were treated with 1560 M ursolic acid, it inhibited cell viability in a dose-dependent manner, increased caspase-3, p-JNK/t-JNK expression, and reduced VEGF levels