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Gut dysbiosis: ecological causes and causative effects on human disease

Current studies suggest that the pathophysiological mechanisms of EDC involve multiple factors, including chronic inflammation, neurotransmitter imbalance, hypothalamicpituitaryadrenal (HPA) axis dysfunction, and impaired synaptic plasticity (Yan et al., 2025)

Overexpression of TFRC contributes to the continuous accumulation of ferrous iron and autophagic lysosome-mediated degradation of ferritin, and promotes lipid peroxidation and cell collapse, resulting in ferroptosis-related cell death [37, 38]

In this model, CRTC1 was identified as a direct target of miR-184-3p, suggesting that the loss of this miRNA compromises molecular mechanisms essential for HPA axis homeostasis and affective behavior regulation (Xu et al., 2024)