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In this study, both AOD9604 and HA individually led to improved cartilage outcomes and shorter periods of lameness compared to saline control

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tumor necrosis factor (TNF)-, interleukin (IL)-17, IL-22, IL-23) produced by immune cells that infiltrate the dermis, leading to epidermal hyperproliferation via activation of signal transducer and activator of transcription 3 (STAT3) 2