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Drug design challenges Several issues must be addressed concerning the development of an efficacious drug to treat AD: (1) the drug must penetrate the BBB to influence damaged neurons in memory-associated brain structures

Conventional copper-modulating therapies, including chelators (e.g., TTM and TETA) and the ionophore elesclomol, are fundamentally limited by their lack of specificity, which constrains their clinical utility and leads to significant adverse effects

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