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We emphasise the need to characterise repetitive and transposable elements and understand their influence on expression mechanisms of genes biologically implicated in AN

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166 ROS promote extrinsic apoptosis by accelerating the ubiquitin-mediated degradation of c-FLIP, thereby enhancing DISC formation and caspase-8 activation through adapter proteins such as Fas-associated death domain (FADD) and TNF receptor-associated death domain (TRADD)