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For instance, these cells identify melanocytes via Major Histocompatibility Complex (MHC) class I molecules and induce apoptosis through the directed release of perforin and granzyme B, while IFN- secretion exacerbates local inflammation and amplifies the autoimmune response, constituting a key pathological event in vitiligo ( Recent research highlights that environmental factors, such as ultraviolet (UV) radiation or chemical exposure, can trigger vitiligo by inducing the production of pathogen-associated molecular patterns (PAMPs) or damage-associated molecular patterns (DAMPs) ( + T cells target melanocytes, resulting in skin depigmentation

Precaution: A twice-yearly full body skin examination is recommended for patients using the approved brand Scenesse, as there is an ongoing concern that increased melanocyte proliferation may elevate the risk of atypical moles or melanoma in susceptible individuals

The agitation is too violent for these long-chain amino acids