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Description

beta-Amyloid disrupts activity-dependent gene transcription required for memory through the CREB coactivator CRTC1

Toxicity of DCVC to kidney cells has been associated with (1) covalent modification of macromolecules, (2) depletion of nonprotein thiols (presumably mostly GSH), (3) lipid peroxidation (Beuter et al., 1989, Chen et al., 1990, Groves et al., 1991), and (4) carbonylation of susceptible proteins (Fan et al., 2014)