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Using a well-established model of oxidatively damaged IVF mouse embryos, we applied the iTRAQ method to identify proteins differentially expressed between control and oxidatively damaged zygotes and explored the possible tumorigenic mechanisms, especially with regard to the effects of oxidative damage on ribosome biogenesis closely related to tumorigenesis

The insight most users miss: IGF-1 LR3's extended half-life (2030 hours vs 10 minutes for endogenous IGF-1) means systemic circulation, not localized muscle activation