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This study demonstrated that GES had potential to influence CNS control mechanisms related to nausea and vomiting via activation of vagal afferent pathway, and then increased vagal efferent tone to mitigate gastric sensitivity, all of which ameliorated postprandial gastric accommodation

Similarly, transcripts for Sox2 , Mist1, and Aqp5 were reduced immediately following IR but returned to control levels by day 7 postIR (Fig EV4B)

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