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Curr.Drug Metab 2006;7(7):811-816

761 The overexpression of CYP1B1 increases the ubiquitination and degradation of ACSL4, and melatonin could inhibit ferroptosis by regulating the ACSL4/CYP1B1 pathway, significantly reducing the cerebral ischemic area and neuron loss in MCAO mice

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Under the specified temperature, pH, and time conditions, the substrate can undergo thorough enzymatic hydrolysis, facilitating the more efficient removal of starch and protein