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The authors show that the drastic growth arrest of the sir1 mutant is partially rescued when glutathione deficiency is introduced via the cad2 mutation, which decreases glutathione levels to 30% to 40% of wild-type levels (Cobbett et al., 1998), or pharmacologically through treatment with the GCL inhibitor buthionine sulfoximine

Liposomal delivery encapsulates the reduced glutathione molecule inside microscopic phospholipid vesicles (liposomes) that mirror the structure of cell membranes

doi:10.1152/japplphysiol.00945.2010