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ChemBioChem 2022, 23, e202100514

EVs of ADSCs origin up-regulated the gene expression of glutamine synthetase in hepatocytes while down-regulating the expression of GLS2, which interfered with glutamine metabolism as well as the progression of hepatic fibrosis [129]

Experimental and translational studies suggest that elevated prolactin levels contribute to obesity-related phenotypes through multiple pathways, including altered central appetite regulation, modulation of adipocyte differentiation and lipid storage, impairment of insulin sensitivity, and disruption of the hypothalamicpituitarygonadal (HPG) axis