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Conclusions In this study, 16S rRNA sequencing, metabolomics, metabolite traceability, and molecular docking techniques were integrated to demonstrate that there was a cross-talk between gastric mucosal microbes and mitochondria

At 6 weeks of age, mice were housed in plastic cages and received standard chow (AIN-76) and water ad libitum prior to experiment maintained on a 12 h/12 h light/dark cycle

We further demonstrated that an increase in urinary GSH concentrations during the formation of TLS is a common phenomenon in both mice and humans and proposed the utility of urinary GSH as a biomarker to detect TLS in the kidney