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As shown in the result, it was the ferroptosis inhibitors (ferrostatin-1, Deferoxamine, N-Acetyl-L-cysteine) that restored cell viability but not other cell death inhibitors, including Bafilomycin A1 (autophagy inhibitor), Z-VAD-FMK (apoptosis inhibitor), Necrosulfonamide (necroptosis inhibitor) (Figs

In the animal model [105], lutein and zeaxanthin were found to modulate anti-inflammatory cytokine (IL-10) in the liver and jejunum as well as proinflammatory cytokines (IL-1, IL-6, and IFN-) in the liver, duodenum, jejunum, and ileum

References Brockway DF, Crowley NA

Enhanced Keap1-Nrf2 signaling protects the myocardium from isoproterenol-induced pathological remodeling in mice