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[373] claimed that chlorogenic acid is able to reduce DSS-induced colonic mucosal damage, by alleviating DSS-induced inflammation, oxidative stress, and apoptosis in the colon, while reducing ERK1/2, p -ERK, p38, p-p38, JNK, and p-JNK protein expression

Similarly, the results of our present study demonstrate that catechol estrogens can strongly protect cells against chemically-induced ferroptosis through inhibition of PDI and subsequent abrogation of cellular NO, ROS and lipid-ROS accumulation 36

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But for those with certain liver enzyme differences or microbiomes that favor TMA production, the risk is tangible and manageable