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Hyperglycemia, dyslipidemia, and nicotine can elevate ROS levels in ECs [43], upregulating high mobility group box 1(HMGB1) and potentiating pyroptosis owing to the inflammasome

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As central regulatory nodes linking glycolysis and mitochondrial metabolism, PDKs act as critical metabolic gatekeepers that orchestrate metabolic plasticity and modulate cellular adaptation to cisplatin-induced stress, ultimately influencing the therapeutic response [37]