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Animal models confirm TMAOs pathogenic role: reducing plasma TMAO slows CKD progression (138), while TMAO accelerates diabetic nephropathy by activating NLRP3 inflammasome, promoting IL-1/IL-18 release, and worsening renal dysfunction/fibrosis (139)

The researchers hypothesized that one or more medications, called antagonists, could inhibit the damaging signaling caused by TGF-B and PI3K/AKT/mTOR/p70 S6K, which converts the VICs from healthy to damaged

Increased citrullination is not specific to RA and occurs regularly with any environmental stress, including in lung alveolar macrophages in cigarette smokers (6)