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The first phase revealed that peptide hormones can be subject to processing, and that their pro-forms may involve regulatory stages

Treatment with the protein glycosylation inhibitor tunicamycin verified reduction of the ~100 kDa glycosylated CD98HC with concomitant increase in ~5060 kDa unglycosylated CD98HC (Fig

Additionally, alanine mutations of E124 3.33 and R283 6.55 also lead to the significantly diminished basal activity of the ghrelin receptor, indicating a potential role of the salt bridge formed by these residues in modulating the receptors basal activity (Supplementary Fig

Oxidative stress in inflammation-based gastrointestinal tract diseases: challenges and opportunities