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glutathione neurotransmitters

glutathione neurotransmitters Controlling entry into mitochondria: potential roles for SLC25A39 in health and (treatment of) disease GLUTAMATE ABNORMALITIES IN OBSESSIVE COMPULSIVE

GLUTAMATE ABNORMALITIES IN OBSESSIVE COMPULSIVE DISORDER: NEUROBIOLOGY, PATHOPHYSIOLOGY, AND TREATMENT PMC Glutathione in Brain Disorders and Aging PMC Current advances in the detection of neurotransmitters by nanomaterials: An update ScienceDirect Amino Acids as Neurotransmitters. The Balance between Excitation and Inhibition as a Background for Future Clinical Applications IntechOpen

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Cobalamin deficiency results in the decrease of this conversion, resulting in high plasma levels of homocysteine, which is associated with a higher risk of cerebrovascular disease, depression, dementia, cellular stress, and apoptosis (, , , )

glutathione neurotransmitters Controlling entry into mitochondria: potential roles for SLC25A39 in health and (treatment of) disease GLUTAMATE ABNORMALITIES IN OBSESSIVE COMPULSIVE

70 As cells use NADPH to reduce cystine to cysteine, the increased cysteine catabolism by CDO implies the increased consumption of NADPH, which will affect other cell metabolic pathways and the antioxidant capacity as well

glutathione neurotransmitters Controlling entry into mitochondria: potential roles for SLC25A39 in health and (treatment of) disease GLUTAMATE ABNORMALITIES IN OBSESSIVE COMPULSIVE

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glutathione neurotransmitters Controlling entry into mitochondria: potential roles for SLC25A39 in health and (treatment of) disease GLUTAMATE ABNORMALITIES IN OBSESSIVE COMPULSIVE

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glutathione neurotransmitters Controlling entry into mitochondria: potential roles for SLC25A39 in health and (treatment of) disease GLUTAMATE ABNORMALITIES IN OBSESSIVE COMPULSIVE
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