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Immediately before each measurement, cells were treated with rotenone (10 M) to enhance the production of mitochondrial ROS

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In the case of preeclampsia, currently, the metabolic investigations center on placental energy and metabolic intermediates, the alterations of which are caused by metabolic reprogramming, ultimately leading to insufficient placental energy and perturbation of nutrient synthesis

Special emphasis will be put on the ferroptosiss vital role in EOC, primarily covering its role in tumorigenesis and progression of EOC, the capability of reversing chemotherapy resistance, and the research and development of related therapeutic strategies