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Since the capacity of GR1 in the Arabidopsis cytosol is large and far from limiting under normal growth conditions 63 , inhibition of GR1 by (hyper)oxidation of catalytic or regulatory cysteine residues provide a plausible hypothesis 64 either as a result of oxidative damage or as a mechanism to safeguard NADP redox status, similar to what has previously been observed for peroxiredoxins 50,65

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