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Description

G., Sharma, S

In HCECs applied with desiccating stress, the CEC ferroptosis was triggered by the aggregation of autophagy impairment-derived sequestosome 1 (SQSTM1), the overexpression of which fostered ferroptotic changes and substantially elevated ACSL4 (124)

The IL-6/IL-6R signal promotes the formation of an immune-inflammatory microenvironment by inducing KP activation in pulmonary vascular cells (Cai et al., 2022)

GHK-Cu has been shown to accelerate wound closure, reduce scarring, and promote the formation of new blood vessels (angiogenesis) crucial for tissue repair