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These aggressive protocols typically run shorter durations

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Other studies indicate that while stress may play a part, it is only a small part of a bigger picture where disease and other factors contribute

Finally, unbiased phosphoproteomics combined with kinase inhibition has revealed hundreds of novel GSK3 substrates [34], many of which have known or plausible roles in GLUT4 traffic including the regulator of GLUT4 endocytosis GAPVD1/GAPEX5 (GSK3 sites: S758 and T762 in mice) [106108], the early/recycling endosome regulator RBSN/Rabenosyn-5 (S225) [109], and the endosome-to-Golgi trafficking regulator GOLGA4/golgin-245 (T39 and S40) [110]