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A recent work has shown that epileptic seizures lead to overexpression of Keap1 promoting proteasomal degradation of NRF-2 in neurons, while inhibition of Keap1 expression stabilizes NRF-2, and increases the level of GSH, GPX4, superoxide dismutase (SOD), and the cystine/glutamate exchanger subunit SLC7A11 [47]

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Pathway enrichment analysis, conducted using STRING-DB, revealed that the most significantly enriched pathways were associated with microbial metabolism in diverse environments, ABC transporters, two-component systems, chemotaxis, sulfur metabolism, flagellar assembly, etc

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