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Glutathione plays a pivotal role in most key physiological functions, including but not limited to maintenance of cellular redox, neutralising free radicals, cell cycle regulation, proliferation, apoptosis, xenobiotic metabolism, and the recycling of other cellular antioxidants such as Vitamins C and E

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Given the close interactions among stress, GC signaling, and hippocampal neurogenesis, ME/CFS-related conditions characterized by chronic stress exposure and HPA-axis dysregulation may plausibly involve vulnerability in neurogenic processes and related plasticity networks