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[27] Androgen receptor is modified by post-translational modification through acetylation, [28] which directly promotes AR-mediated transactivation, apoptosis [29] and contact-independent growth of prostate cancer cells

Hypoxic regulation of glutamine metabolism through HIF1 and SIAH2 supports lipid synthesis that is necessary for tumor growth

Modification of severe insulin resistant diabetes in response to lifestyle changes in Alstrom syndrome

Divergences from the consensus composition of an AR DNA-binding motif appeared to be associated also with the stage of CaP progression, as changes in ARE composition and location are seen both during prostate carcinogenesis and the development of ADT resistance