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Moreover, infiltrating T cells upregulate Nox4 in the inflammatory microenvironment and produce a large number of extracellular ROS, such that they themselves undergo ferroptosis, thereby weakening immune surveillance

In the future, many issues need to be clarified in more epigenetic molecules and detailed mechanistic insights to design the effective cancer therapy strategies based on ferroptosis

Overexpression of EMP1 potentiates RSL3-mediated GPX4 suppression, thereby induces ferroptosis