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(January 2000)

As mentioned before, there is current clinical interest in using low dose estradiol therapy to treat antihormone resistant breast cancer [24] however only a minimal 30% of patients respond to this therapeutic strategy

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After analyzing blood and tumor samples that had been collected over time from some participants, one finding of particular interest was that treatment with atezolizumab could "convert" tumors from those that initially lacked one of two proteins, PD-1 or PD-L1, needed for atezolizumab to work, to those that eventually produced high levels of either protein