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Additionally, TDP-43 with the G348C mutation forms significantly larger stress granules, and is incorporated into the stress granules earlier than the wild-type TDP-43, although eventually, the wild-type TDP-43 expressing cells form more number of stress granules per cell, albeit, the granule sizes remain unchanged (Dewey et al., 2011)

Poly (ADP-Ribose) polymerase 1 (PARP1), as an enzyme crucial for repairing DNA damage, can effectively repair damaged DNA by mediating abnormal activation of the base excision repair (BER) pathway, thereby resulting in oxaliplatin resistance (47)

Membrane perforins are characteristic features of other cell death modalities, such as BAX/BAK in apoptosis [10], MLKL in necroptosis [11], GSDM and NINJ1 in pyroptosis [12,13,14,15], however this is not the case for ferroptosis

PMID: 28448947 Drucker DJ (2022): GLP-1 physiology informs the pharmacotherapy of obesity