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Mice lacking both isoforms are resistant to diet-induced obesity, but either die of severe hypoglycemia shortly after birth or live with severe metabolic imbalances ( Cebp / animals restores adiposity and lipid homeostasis, including plasma leptin, free fatty acids, and triglycerides levels ( Beginning from a geroscience framework, we showed that reduced mTORC1 signaling prevents diet-induced obesity by increasing the activity of C/EBP-

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