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While these are promising strategies, it is essential that the suggested effects of loss-of-function ATXN3 [71] are well studied in order to evaluate if the beneficial effects outweigh the risks, particularly because ATXN3 gene silencing in some cell types has been shown to cause accumulation of DNA damage, increased proliferation, and loss of adhesion and of differentiation, all of which are hallmarks of cancer [71, 282]

It was first characterized by Professor Predrag Sikiric and colleagues at the University of Zagreb in the early 1990s

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