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Experimental models demonstrate that high-dose testosterone administration increases hepatic reactive oxygen species production while suppressing endogenous antioxidant enzyme activity ( Intrahepatic cholestasis observed in anabolic steroid users has been linked to impaired bile-acid transport

These phases act in concert to detoxify drugs and remove them from cells and eventually from the body

It stimulates the production of precursors necessary for glutathione synthesis and induces Nrf2 release into the nucleus