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Interestingly, both genistein and 17-estradiol treatment significantly reduced the number of apoptotic neurons, as well as the number of neurons expressing active caspase-3, a key executioner of programmed cell death

as well as CAT activity [34]

Nature Commun 6 , 8265 (2015)

Understanding how these chaperone systems are coordinated and how their protective capacity declines with age and disease will be essential for developing effective proteostasis-based therapies for NDDs